Molecular mechanisms involved in NAFLD progression - PubMed
Review
Molecular mechanisms involved in NAFLD progression
Mariano Malaguarnera et al. J Mol Med (Berl). 2009 Jul.
Abstract
Non-alcoholic fatty liver disease (NAFLD) is an emerging metabolic-related disorder characterized by fatty infiltration of the liver in the absence of alcohol consumption. NAFLD ranges from simple steatosis to non-alcoholic steatohepatitis (NASH), which might progress to end-stage liver disease. This progression is related to the insulin resistance, which is strongly linked to the metabolic syndrome consisting of central obesity, diabetes mellitus, and hypertension. Earlier, the increased concentration of intracellular fatty acids within hepatocytes leads to steatosis. Subsequently, multifactorial complex interactions between nutritional factors, lifestyle, and genetic determinants promote necrosis, inflammation, fibrosis, and hepatocellular damage. Up to now, many studies have revealed the mechanism associated with insulin resistance, whereas the mechanisms related to the molecular components have been incompletely characterized. This review aims to assess the potential molecular mediators initiating and supporting the progression of NASH to establish precocious diagnosis and to plan more specific treatment for this disease.
Similar articles
-
Non-alcoholic steatohepatitis: pathogenesis and novel therapeutic approaches.
Schuppan D, Schattenberg JM. Schuppan D, et al. J Gastroenterol Hepatol. 2013 Aug;28 Suppl 1:68-76. doi: 10.1111/jgh.12212. J Gastroenterol Hepatol. 2013. PMID: 23855299 Review.
-
[Non-alcoholic fatty liver disease--new view].
Raszeja-Wyszomirska J, Lawniczak M, Marlicz W, Miezyńska-Kurtycz J, Milkiewicz P. Raszeja-Wyszomirska J, et al. Pol Merkur Lekarski. 2008 Jun;24(144):568-71. Pol Merkur Lekarski. 2008. PMID: 18702346 Review. Polish.
-
Gerges SH, Wahdan SA, Elsherbiny DA, El-Demerdash E. Gerges SH, et al. Life Sci. 2021 Apr 15;271:119220. doi: 10.1016/j.lfs.2021.119220. Epub 2021 Feb 13. Life Sci. 2021. PMID: 33592199 Review.
-
Molecular mechanisms of steatosis in nonalcoholic fatty liver disease.
Pettinelli P, Obregón AM, Videla LA. Pettinelli P, et al. Nutr Hosp. 2011 May-Jun;26(3):441-50. doi: 10.1590/S0212-16112011000300003. Nutr Hosp. 2011. PMID: 21892559 Review.
-
Fatty liver: role of inflammation and fatty acid nutrition.
Byrne CD. Byrne CD. Prostaglandins Leukot Essent Fatty Acids. 2010 Apr-Jun;82(4-6):265-71. doi: 10.1016/j.plefa.2010.02.012. Epub 2010 Mar 2. Prostaglandins Leukot Essent Fatty Acids. 2010. PMID: 20189787 Review.
Cited by
-
Miyake T, Miyazaki M, Yoshida O, Kanzaki S, Nakaguchi H, Nakamura Y, Watanabe T, Yamamoto Y, Koizumi Y, Tokumoto Y, Hirooka M, Furukawa S, Takeshita E, Kumagi T, Ikeda Y, Abe M, Toshimitsu K, Matsuura B, Hiasa Y. Miyake T, et al. BMC Gastroenterol. 2021 Apr 13;21(1):170. doi: 10.1186/s12876-021-01748-y. BMC Gastroenterol. 2021. PMID: 33849437 Free PMC article.
-
Caballero F, Fernández A, Matías N, Martínez L, Fucho R, Elena M, Caballeria J, Morales A, Fernández-Checa JC, García-Ruiz C. Caballero F, et al. J Biol Chem. 2010 Jun 11;285(24):18528-36. doi: 10.1074/jbc.M109.099333. Epub 2010 Apr 15. J Biol Chem. 2010. PMID: 20395294 Free PMC article.
-
Treating nonalcoholic steatohepatitis with antidiabetic drugs: Will GLP-1 agonists end the struggle?
Kalogirou M, Sinakos E. Kalogirou M, et al. World J Hepatol. 2018 Nov 27;10(11):790-794. doi: 10.4254/wjh.v10.i11.790. World J Hepatol. 2018. PMID: 30533179 Free PMC article.
-
Role of mitochondria in nonalcoholic fatty liver disease.
Nassir F, Ibdah JA. Nassir F, et al. Int J Mol Sci. 2014 May 15;15(5):8713-42. doi: 10.3390/ijms15058713. Int J Mol Sci. 2014. PMID: 24837835 Free PMC article. Review.
-
Molecular and cellular mechanisms of liver fibrosis and its regression.
Kisseleva T, Brenner D. Kisseleva T, et al. Nat Rev Gastroenterol Hepatol. 2021 Mar;18(3):151-166. doi: 10.1038/s41575-020-00372-7. Epub 2020 Oct 30. Nat Rev Gastroenterol Hepatol. 2021. PMID: 33128017 Review.
References
Publication types
MeSH terms
LinkOut - more resources
Full Text Sources
Other Literature Sources